Alberto Lleó Bisa-rekin lankidetzan egindako argitalpenak (10)

2023

  1. Erratum: Author Correction: Common variants in Alzheimer's disease and risk stratification by polygenic risk scores (Nature communications (2021) 12 1 (3417))

    Nature communications

2022

  1. Intermediate and Expanded HTT Alleles and the Risk for α-Synucleinopathies

    Movement Disorders, Vol. 37, Núm. 9, pp. 1841-1849

  2. New insights into the genetic etiology of Alzheimer's disease and related dementias

    Nature genetics, Vol. 54, Núm. 4, pp. 412-436

2021

  1. Common variants in Alzheimer's disease and risk stratification by polygenic risk scores

    Nature communications, Vol. 12, Núm. 1, pp. 3417

  2. Genetic variation in APOE, GRN, and TP53 are phenotype modifiers in frontotemporal dementia

    Neurobiology of Aging, Vol. 99, pp. 99.e15-99.e22

  3. Long runs of homozygosity are associated with Alzheimer’s disease

    Translational Psychiatry, Vol. 11, Núm. 1

2020

  1. C9orf72, age at onset, and ancestry help discriminate behavioral from language variants in FTLD cohorts

    Neurology, Vol. 95, Núm. 24, pp. E3288-E3302

  2. Role for ATXN1, ATXN2, and HTT intermediate repeats in frontotemporal dementia and Alzheimer's disease

    Neurobiology of Aging, Vol. 87, pp. 139.e1-139.e7

2019

  1. Genome-wide association analysis of dementia and its clinical endophenotypes reveal novel loci associated with Alzheimer's disease and three causality networks: The GR@ACE project

    Alzheimer's and Dementia, Vol. 15, Núm. 10, pp. 1333-1347

  2. HTT gene intermediate alleles in neurodegeneration: evidence for association with Alzheimer's disease

    Neurobiology of Aging, Vol. 76, pp. 215.e9-215.e14