Publications by the researcher in collaboration with SANTIAGO JESUS CAL MIGUEL (14)

2024

  1. Lung Inflammatory Phenotype in Mice Deficient in Fibulin-2 and ADAMTS-12

    International Journal of Molecular Sciences, Vol. 25, Núm. 4

2021

  1. ADAMTS-12: Functions and Challenges for a Complex Metalloprotease

    Frontiers in Molecular Biosciences, Vol. 8

  2. Hyalectanase activities by the adamts metalloproteases

    International Journal of Molecular Sciences, Vol. 22, Núm. 6, pp. 1-22

2020

  1. New Insights into ADAMTS Metalloproteases in the Central Nervous System

    Biomolecules, Vol. 10, Núm. 3

2019

  1. Antitumor potential of fibulin-5 in breast cancer cells depends on its RGD cell adhesion motif

    Cellular Physiology and Biochemistry, Vol. 53, Núm. 1, pp. 87-100

  2. Neurocan is a new substrate for the ADAMTS12 metalloprotease: Potential implications in neuropathies

    Cellular Physiology and Biochemistry, Vol. 52, Núm. 5, pp. 1003-1016

  3. Novel associations within the tumor microenvironment: Fibulins meet ADAMTSs

    Frontiers in Oncology, Vol. 9, Núm. AUG

  4. The molecular interaction of ADAMTS-1 and fibulin-1 and its potential contribution to breast cancer biology

    Journal of Cancer Metastasis and Treatment, Vol. 5

2018

  1. Periostin, dentin matrix protein 1 and P2rx7 ion channel in human teeth and periodontal ligament

    Annals of Anatomy, Vol. 216, pp. 103-111

2016

  1. Role of periostin in adhesion and migration of bone remodeling cells

    PLoS ONE, Vol. 11, Núm. 1

2015

  1. Immunohistochemical localization of periostin in human gingiva

    European Journal of Histochemistry, Vol. 59, Núm. 3, pp. 207-210

2013

  1. Cell-cell adhesion genes CTNNA2 and CTNNA3 are tumour suppressors frequently mutated in laryngeal carcinomas

    Nature Communications, Vol. 4

2009

  1. Genetic inactivation of ADAMTS15 metalloprotease in human colorectal cancer

    Cancer Research, Vol. 69, Núm. 11, pp. 4926-4934

2007

  1. Human DESC1 serine protease confers tumorigenic properties to MDCK cells and it is upregulated in tumours of different origin

    British Journal of Cancer, Vol. 97, Núm. 2, pp. 201-209