Bioquímica y Biología Molecular
Departamento
Centro Nacional de Investigaciones Cardiovasculares Carlos III
Madrid, EspañaPublicaciones en colaboración con investigadores/as de Centro Nacional de Investigaciones Cardiovasculares Carlos III (38)
2024
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Endothelial-to-Mesenchymal Transition Contributes to Accelerated Atherosclerosis in Hutchinson-Gilford Progeria Syndrome
Circulation
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Exacerbated atherosclerosis in progeria is prevented by progerin elimination in vascular smooth muscle cells but not endothelial cells
Proceedings of the National Academy of Sciences of the United States of America, Vol. 121, Núm. 18
2023
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Intravenous administration of BCG in mice promotes natural killer and T cell-mediated antitumor immunity in the lung
Nature communications, Vol. 14, Núm. 1, pp. 6090
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USP49 deubiquitinase regulates the mitotic spindle checkpoint and prevents aneuploidy
Cell Death and Disease, Vol. 14, Núm. 1
2022
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Autophagy-linked plasma and lysosomal membrane protein PLAC8 is a key host factor for SARS-CoV-2 entry into human cells
EMBO Journal, Vol. 41, Núm. 21
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Isolation of Mouse Aortic RNA for Transcriptomics
Methods in molecular biology (Clifton, N.J.), Vol. 2419, pp. 611-627
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Vascular smooth muscle cell aging: Insights from Hutchinson-Gilford progeria syndrome
Clinica e Investigacion en Arteriosclerosis
2021
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Cardiovascular Progerin Suppression and Lamin A Restoration Rescue Hutchinson-Gilford Progeria Syndrome
Circulation, Vol. 144, Núm. 22, pp. 1777-1794
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Genomic instability in the naturally and prematurely aged myocardium
Proceedings of the National Academy of Sciences of the United States of America, Vol. 118, Núm. 36
2020
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Biological Versus Chronological Aging: JACC Focus Seminar
Journal of the American College of Cardiology, Vol. 75, Núm. 8, pp. 919-930
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Identification of common cardiometabolic alterations and deregulated pathways in mouse and pig models of aging
Aging Cell, Vol. 19, Núm. 9
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Premature Vascular Aging with Features of Plaque Vulnerability in an Atheroprone Mouse Model of Hutchinson-Gilford Progeria Syndrome with Ldlr Deficiency
Cells, Vol. 9, Núm. 10
2019
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Analysis of How Emergency Physicians’ Decisions to Hospitalize or Discharge Patients With Acute Heart Failure Match the Clinical Risk Categories of the MEESSI-AHF Scale
Annals of Emergency Medicine, Vol. 74, Núm. 2, pp. 204-215
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Progerin accelerates atherosclerosis by inducing endoplasmic reticulum stress in vascular smooth muscle cells
EMBO Molecular Medicine, Vol. 11, Núm. 4
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Remodeling of Bone Marrow Hematopoietic Stem Cell Niches Promotes Myeloid Cell Expansion during Premature or Physiological Aging
Cell Stem Cell, Vol. 25, Núm. 3, pp. 407-418.e6
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Vascular smooth muscle cell loss underpins the accelerated atherosclerosis in Hutchinson-Gilford progeria syndrome
Nucleus, Vol. 10, Núm. 1, pp. 28-34
2018
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Ablation of the stress protease OMA1 protects against heart failure in mice
Science Translational Medicine, Vol. 10, Núm. 434
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Accelerated atherosclerosis in HGPS
Aging
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Aging in the Cardiovascular System: Lessons from Hutchinson-Gilford Progeria Syndrome
Annual Review of Physiology, Vol. 80, pp. 27-48
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Mechanisms of vascular aging: What can we learn from Hutchinson-Gilford progeria syndrome?
Clinica e Investigacion en Arteriosclerosis