Publications by the researcher in collaboration with Vicente Andrés García (24)

2024

  1. Endothelial-to-Mesenchymal Transition Contributes to Accelerated Atherosclerosis in Hutchinson-Gilford Progeria Syndrome

    Circulation

  2. Exacerbated atherosclerosis in progeria is prevented by progerin elimination in vascular smooth muscle cells but not endothelial cells

    Proceedings of the National Academy of Sciences of the United States of America, Vol. 121, Núm. 18

2022

  1. Isolation of Mouse Aortic RNA for Transcriptomics

    Methods in molecular biology (Clifton, N.J.), Vol. 2419, pp. 611-627

2021

  1. Cardiovascular Progerin Suppression and Lamin A Restoration Rescue Hutchinson-Gilford Progeria Syndrome

    Circulation, Vol. 144, Núm. 22, pp. 1777-1794

  2. Genomic instability in the naturally and prematurely aged myocardium

    Proceedings of the National Academy of Sciences of the United States of America, Vol. 118, Núm. 36

2020

  1. Biological Versus Chronological Aging: JACC Focus Seminar

    Journal of the American College of Cardiology, Vol. 75, Núm. 8, pp. 919-930

  2. Identification of common cardiometabolic alterations and deregulated pathways in mouse and pig models of aging

    Aging Cell, Vol. 19, Núm. 9

  3. Premature Vascular Aging with Features of Plaque Vulnerability in an Atheroprone Mouse Model of Hutchinson-Gilford Progeria Syndrome with Ldlr Deficiency

    Cells, Vol. 9, Núm. 10

2019

  1. Progerin accelerates atherosclerosis by inducing endoplasmic reticulum stress in vascular smooth muscle cells

    EMBO Molecular Medicine, Vol. 11, Núm. 4

  2. Remodeling of Bone Marrow Hematopoietic Stem Cell Niches Promotes Myeloid Cell Expansion during Premature or Physiological Aging

    Cell Stem Cell, Vol. 25, Núm. 3, pp. 407-418.e6

  3. Vascular smooth muscle cell loss underpins the accelerated atherosclerosis in Hutchinson-Gilford progeria syndrome

    Nucleus, Vol. 10, Núm. 1, pp. 28-34

2018

  1. Accelerated atherosclerosis in HGPS

    Aging

  2. Aging in the Cardiovascular System: Lessons from Hutchinson-Gilford Progeria Syndrome

    Annual Review of Physiology, Vol. 80, pp. 27-48

  3. Mechanisms of vascular aging: What can we learn from Hutchinson-Gilford progeria syndrome?

    Clinica e Investigacion en Arteriosclerosis

  4. The microRNA-29/PGC1α regulatory axis is critical for metabolic control of cardiac function

    PLoS Biology, Vol. 16, Núm. 10

  5. Vascular smooth muscle–specific progerin expression accelerates atherosclerosis and death in a mouse model of Hutchinson-Gilford progeria syndrome

    Circulation, Vol. 138, Núm. 3, pp. 266-282

2017

  1. Novel phosphate-activated macrophages prevent ectopic calcification by increasing extracellular ATP and pyrophosphate

    PLoS ONE, Vol. 12, Núm. 3

2016

  1. Alternatively activated macrophages exhibit an anticalcifying activity dependent on extracellular ATP/pyrophosphate metabolism

    American Journal of Physiology - Cell Physiology, Vol. 310, Núm. 10, pp. C788-C799

  2. Cardiac electrical defects in progeroid mice and Hutchinson-Gilford progeria syndrome patients with nuclear lamina alterations

    Proceedings of the National Academy of Sciences of the United States of America, Vol. 113, Núm. 46, pp. E7250-E7259

2015

  1. In vitro macrophage phagocytosis assay

    Methods in Molecular Biology (Humana Press Inc.), pp. 235-246