Publications in collaboration with researchers from University of Queensland (13)

2023

  1. A second update on mapping the human genetic architecture of COVID-19

    Nature

  2. Erratum to: Mapping the human genetic architecture of COVID-19 (Nature, (2021), 600, 7889, (472-477), 10.1038/s41586-021-03767-x)

    Nature

2022

  1. Whole-genome sequencing reveals host factors underlying critical COVID-19

    Nature, Vol. 607, Núm. 7917, pp. 97-103

2021

  1. Guidelines for the use and interpretation of assays for monitoring autophagy (4th edition)1

    Autophagy, Vol. 17, Núm. 1, pp. 1-382

2019

  1. Publisher Correction: Common schizophrenia alleles are enriched in mutation-intolerant genes and in regions under strong background selection (Nature Genetics, (2018), 50, 3, (381-389), 10.1038/s41588-018-0059-2)

    Nature Genetics

2018

  1. Common schizophrenia alleles are enriched in mutation-intolerant genes and in regions under strong background selection

    Nature Genetics, Vol. 50, Núm. 3, pp. 381-389

2016

  1. Erratum to: Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition) (Autophagy, 12, 1, 1-222, 10.1080/15548627.2015.1100356

    Autophagy

  2. Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

    Autophagy, Vol. 12, Núm. 1, pp. 1-222

2015

  1. ERAP2 is associated with ankylosing spondylitis in HLA-B27-positive and HLA-B27-negative patients

    Annals of the Rheumatic Diseases

  2. Major histocompatibility complex associations of ankylosing spondylitis are complex and involve further epistasis with ERAP1

    Nature Communications, Vol. 6

  3. Systems Biology Approaches to Understand Natural Products Biosynthesis

    Frontiers in Bioengineering and Biotechnology, Vol. 3

2013

  1. Identification of multiple risk variants for ankylosing spondylitis through high-density genotyping of immune-related loci

    Nature Genetics, Vol. 45, Núm. 7, pp. 730-738

2011

  1. Interaction between ERAP1 and HLA-B27 in ankylosing spondylitis implicates peptide handling in the mechanism for HLA-B27 in disease susceptibility

    Nature Genetics, Vol. 43, Núm. 8, pp. 761-767