Publications in collaboration with researchers from University of Pittsburgh Cancer Institute (11)

2020

  1. Protein-altering germline mutations implicate novel genes related to lung cancer development

    Nature Communications, Vol. 11, Núm. 1

2019

  1. Genetic interaction analysis among oncogenesis-related genes revealed novel genes and networks in lung cancer development

    Oncotarget, Vol. 10, Núm. 19, pp. 1760-1774

  2. Systematic analyses of regulatory variants in DNase i hypersensitive sites identified two novel lung cancer susceptibility loci

    Carcinogenesis, Vol. 40, Núm. 3, pp. 432-440

2018

  1. Fine mapping of MHC region in lung cancer highlights independent susceptibility loci by ethnicity

    Nature Communications, Vol. 9, Núm. 1

  2. Genome-wide interaction study of smoking behavior and non-small cell lung cancer risk in Caucasian population

    Carcinogenesis, Vol. 39, Núm. 3, pp. 336-346

  3. Identification of susceptibility pathways for the role of chromosome 15q25.1 in modifying lung cancer risk

    Nature Communications, Vol. 9, Núm. 1

2017

  1. Large-scale association analysis identifies new lung cancer susceptibility loci and heterogeneity in genetic susceptibility across histological subtypes

    Nature Genetics, Vol. 49, Núm. 7, pp. 1126-1132

2016

  1. Identification of a novel susceptibility locus at 13q34 and refinement of the 20p12.2 region as a multi-signal locus associated with bladder cancer risk in individuals of european ancestry

    Human Molecular Genetics, Vol. 25, Núm. 6, pp. 1203-1214

2014

  1. Genome-wide association study identifies multiple loci associated with bladder cancer risk

    Human Molecular Genetics, Vol. 23, Núm. 5

  2. Imputation and subset-based association analysis across different cancer types identifies multiple independent risk loci in the TERT-CLPTM1L region on chromosome 5p15.33

    Human Molecular Genetics, Vol. 23, Núm. 24, pp. 6616-6633

  3. The 19q12 bladder cancer GWAS signal: Association with cyclin E function and aggressive disease

    Cancer Research, Vol. 74, Núm. 20, pp. 5808-5818