Publicaciones en las que colabora con Clara Soria Valles (15)

2019

  1. Correction: The anti-metastatic activity of collagenase-2 in breast cancer cells is mediated by a signaling pathway involving decorin and miR-21 (Oncogene, (2014), 33, 23, (3054-3063), 10.1038/onc.2013.267)

    Oncogene

  2. Retraction Note: NF-κB activation impairs somatic cell reprogramming in ageing (Nature Cell Biology, (2015), 17, 8, (1004-1013), 10.1038/ncb3207)

    Nature Cell Biology

2017

  1. Immune and inflammatory responses to DNA damage in cancer and aging

    Mechanisms of Ageing and Development, Vol. 165, pp. 10-16

2016

  1. Hallmarks of progeroid syndromes: Lessons from mice and reprogrammed cells

    DMM Disease Models and Mechanisms, Vol. 9, Núm. 7, pp. 719-735

  2. Loss of the proteostasis factor AIRAPL causes myeloid transformation by deregulating IGF-1 signaling

    Nature Medicine, Vol. 22, Núm. 1, pp. 91-96

  3. MMP-25 metalloprotease regulates innate immune response through NF-κB signaling

    Journal of Immunology, Vol. 197, Núm. 1, pp. 296-302

  4. NF-κB signaling as a driver of ageing

    International Review of Cell and Molecular Biology (Elsevier Inc.), pp. 133-174

  5. Novel LMNA mutations cause an aggressive atypical neonatal progeria without progerin accumulation

    Journal of Medical Genetics, Vol. 53, Núm. 11, pp. 776-785

  6. iPSCs: On the Road to Reprogramming Aging

    Trends in Molecular Medicine, Vol. 22, Núm. 8, pp. 713-724

2015

  1. Loss of MT1-MMP causes cell senescence and nuclear defects which can be reversed by retinoic acid

    EMBO Journal, Vol. 34, Núm. 14, pp. 1875-1888

  2. Reprogramming aging through DOT1L inhibition

    Cell Cycle

2014

  1. The anti-metastatic activity of collagenase-2 in breast cancer cells is mediated by a signaling pathway involving decorin and miR-21

    Oncogene, Vol. 33, Núm. 23, pp. 3054-3063

2012

  1. Matrix Proteases and the Degradome

    Matrix Proteases in Health and Disease (Wiley-VCH), pp. 5-23

  2. Nuclear lamina defects cause ATM-dependent NF-κB activation and link accelerated aging to a systemic inflammatory response

    Genes and Development, Vol. 26, Núm. 20, pp. 2311-2324

2011

  1. Analysis of the disintegrin-metalloproteinases family reveals ADAM29 and ADAM7 are often mutated in melanoma

    Human Mutation