Publicaciones en colaboración con investigadores/as de Instituto Universitario de Oncologia del Principado de Asturias (17)

2020

  1. Loss of mitochondrial ClpP, Lonp1, and Tfam triggers transcriptional induction of Rnf213, a susceptibility factor for moyamoya disease

    Neurogenetics, Vol. 21, Núm. 3, pp. 187-203

2019

  1. Global proteome of LonP1+/- mouse embryonal fibroblasts reveals impact on respiratory chain, but no interdependence between eral1 and mitoribosomes

    International Journal of Molecular Sciences, Vol. 20, Núm. 18

  2. Healthspan and lifespan extension by fecal microbiota transplantation into progeroid mice

    Nature Medicine

  3. Methionine restriction for improving progeria: another autophagy-inducing anti-aging strategy?

    Autophagy

  4. Mitochondrial LonP1 protects cardiomyocytes from ischemia/reperfusion injury in vivo

    Journal of Molecular and Cellular Cardiology, Vol. 128, pp. 38-50

2018

  1. Methionine Restriction Extends Lifespan in Progeroid Mice and Alters Lipid and Bile Acid Metabolism

    Cell Reports, Vol. 24, Núm. 9, pp. 2392-2403

  2. Mitohormesis, an Antiaging Paradigm

    International Review of Cell and Molecular Biology, Vol. 340, pp. 35-77

2017

  1. A fruitful liaison of ZSCAN10 and ROS on the road to rejuvenation

    Nature Cell Biology, Vol. 19, Núm. 9, pp. 1012-1013

  2. Autophagy couteracts weight gain, lipotoxicity and pancreatic β-cell death upon hypercaloric pro-diabetic regimens

    Cell Death and Disease, Vol. 8, Núm. 8

  3. Physiological and pathological functions of mitochondrial proteases

    Proteases in Physiology and Pathology (Springer Singapore), pp. 3-25

2016

  1. Interruption of progerin-lamin A/C binding ameliorates Hutchinson-Gilford progeria syndrome phenotype

    Journal of Clinical Investigation, Vol. 126, Núm. 10, pp. 3879-3893

  2. Novel LMNA mutations cause an aggressive atypical neonatal progeria without progerin accumulation

    Journal of Medical Genetics, Vol. 53, Núm. 11, pp. 776-785

2014

  1. ATP-Dependent Lon Protease Controls Tumor Bioenergetics by Reprogramming Mitochondrial Activity

    Cell Reports, Vol. 8, Núm. 2, pp. 542-556

  2. Exome sequencing identifies a novel mutation in PIK3R1 as the cause of SHORT syndrome

    BMC Medical Genetics, Vol. 15, Núm. 1

  3. Lon protease: A key enzyme controlling mitochondrial bioenergetics in cancer

    Molecular and Cellular Oncology, Vol. 1, Núm. 4

2013

  1. Detection of nuclear envelope alterations in senescence

    Methods in Molecular Biology, Vol. 965, pp. 243-251

2012

  1. Nuclear lamina defects cause ATM-dependent NF-κB activation and link accelerated aging to a systemic inflammatory response

    Genes and Development, Vol. 26, Núm. 20, pp. 2311-2324